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Umwelt-Medizin-Gesellschaft
18. Jahrgang, Ausgabe 4/2005, S. 257-340
Die Alzheimer-Krankheit: Quecksilber als pathogener Faktor und Apolipoprotein E als Moderator, J. MUTTER, J. NAUMANN, C. SADAGHIANI, R. SCHNEIDER und H. WALACH
http://www.umwelt-medizin-gesellschaft.de/
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Alzheimererkrankung durch erhöhte Quecksilberbelastung?
Eine erhöhte Quecksilberbelastung kann möglicherweise zu einem Morbus
Alzheimer führen. Dieses Resultat zeigt eine Studie des Instituts für Umweltmedizin und Krankenhaushygiene des Universitätsklinikums Freiburg. Die Wissenschaftler fanden unter anderem heraus, dass aufgrund unterschiedlicher Quecksilberentgiftungskapazitäten einige Menschen besonders
empfindlich auf eine Exposition mit Quecksilber reagieren. Somit könnte eine Quecksilberbelastung bei diesen Personengruppen tatsächlich in ursächlichen Zusammenhang mit der Entstehung eines Morbus Alzheimer gebracht werden. Mittels Blutuntersuchungen sind die Freiburger Ärzte nun in der
Lage, diesen möglicherweise bestehenden Risikofaktor zu ermitteln. In einer weiteren Studie sichten die Forscher derzeit über 1.000 wissenschaftliche Veröffentlichungen zu diesem Themenkomplex. Abstrakt: http://www.neuronews.de/linkto.php?ABOID=31666&ATID=5067&UID=445
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5/2001: Forscher: Handys beschleunigen Alzheimer und Parkinson
Kopenhagen - Handys beschleunigen einige Nervenkrankheiten. Das haben
schwedische Forscher herausgefunden. Nach ihren Erkenntnissen führen die besonderen
Mikrowellen der Mobiltelefone zu Veränderungen im Gehirn. Dadurch werden die
Bluthirn-Barrieren beschädigt. Diese sorgen dafür, dass die´Nervenzellen vor fremden
und giftigen Stoffen geschützt werden. Wenn dieser Schutz wegfällt, werden Krankheiten
wie Alzheimer, Parkinson und Multiple Sklerose beschleunigt. Lesen Sie mehr unter http://www.abendblatt.de/contents/ha/news/allgemeines/html/220501/302205FU15.HTM
Nur in Englisch:
EERC-Led Study Addresses Critical Potential Public Health Risks Related to Pesticide Exposure
July 27, 2006
(GRAND FORKS, NORTH DAKOTA) -- Preliminary results of a project to reduce health risks from pesticide exposure, led by the Energy &
Environmental Research Center (EERC) at the University of North Dakota (UND), verify that exposure to pesticides can induce pathological changes to the nervous system. The pesticide impact program is a strategic effort between the EERC, the UND School of Medicine and Health
Sciences, the College of Nursing, the Psychology Department, and several state and federal agencies.
The EERC team is evaluating how humans are exposed to pesticides so strategies can be developed to reduce health effects for at-risk populations.
"The results of this study are phenomenally relevant to our region and have global implications," said EERC Director Gerald Groenewold. "North Dakota is the perfect laboratory to perform this testing as the state's main industry is agriculture. Airborne pesticides are
more prevalent in our state relative to other classes of pollutants, which makes their effects easier to detect." ....
More at http://www.undeerc.org/newsroom/newsitem.asp?id=267
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Alzheimer's disease with secondary Parkinson's syndrome.
Case report of a patient with dementia and Parkinson's syndrome after long-term occupational exposure to insecticides, herbicides, and pesticides][Article in German]
Laske C,
Wormstall H, Einsiedler K, Buchkremer G. Universitatsklinik fur Psychiatrie und Psychotherapie Tubingen.
Nervenarzt. 2004 Nov;75(11):1107-11.
This case report describes long-term occupational exposure to agricultural insecticides, herbicides, and
pesticides as possible environmental risk factors of Alzheimer's disease (AD) and Parkinson's syndrome in a 59-year-old man. Initially the patient complained about disturbances in concentration, mnestic deficits, and problems finding words. In the further course of the disease, he
developed Parkinson's syndrome with predominant hypokinesia and rigor in addition to mild-to-moderate dementia. Low levels of beta-amyloid 1-42 were found in the CSF. Electroencephalography showed left frontotemporal theta waves. Cranial MRI revealed general brain atrophy with a maximum
biparietally. In cerebral positron emission tomography, general hypometabolism was found with maxima biparietally and left frontally. The possible differential diagnosis of AD and Parkinson's syndrome is discussed.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=abstractplus&list_uids=15551110&query_hl=6&itool=pubmed_docsum
PMID: 15551110 [PubMed - indexed for MEDLINE]
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Uncalculated Risks in Some Pesticides, UCR Study Finds
Some Compounds Show Dramatic Differences in Toxicity And Rate of Break Down Between Isomers
(January 13, 2005)
RIVERSIDE, Calif. –
www.ucr.edu – Researchers at the University of California, Riverside have demonstrated that isomers – or the mirror-image structures – of some pesticides, although chemically identical, have very different biological
and environmental impacts between the two sides. This may have significant implications for risk assessment and research and development directions of new products.
http://www.newsroom.ucr.edu/cgi-bin/display.cgi?id=962&type=print
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Abstract
Annual Review of Public Health Vol. 25: 1-24 (Volume publication date April 2004) (doi:10.1146/annurev.publhealth.25.101802.122951)
First posted online on November 3, 2003
Can
Dementia Be Prevented? Brain Aging in a Population-Based Context
Mary N. Haan and Robert Wallace
University of Michigan, School of Public Health, Epidemiology, Ann Arbor, Michigan 48104;
University of Iowa, Department of Preventive Medicine, Iowa City, Iowa
52240;
As a consequence of global aging of the human population, the occurrence of cognitive impairment and dementia is rapidly becoming a significant burden for medical care and public health systems. By the year 2020, the WHO predicts there will be nearly 29
million demented people in both developed and developing countries. Primary and secondary prevention of dementia through individual and population-level interventions could reduce this imminent risk. Vascular risk factors such as type 2 diabetes, hypertension, dietary fat intake, high
cholesterol, and obesity have emerged as important influences on the risk of both vascular and Alzheimer's dementia. Understanding the reasons for differences between populations in genetic vulnerability and environmental exposures may help to identify modifiable risk factors that may lead
to effective prevention of vascular and Alzheimer's dementia. ...
http://arjournals.annualreviews.org/doi/abs/10.1146/annurev.publhealth.25.101802.122951
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